Aurelia Fraticelli

Aurelia Fraticelli

Dottoranda

PHYS-06/A - Fisica per le scienze della vita, l'ambiente e i beni culturali

aureliafraticelli98@gmail.com

+39 06 72596008

Biografia

Ho conseguito la laurea triennale in Biotecnologia per la salute nel 2022 e la laurea magistrale in Biotecnologie mediche in lingua inglese nel 2024 presso l’Università degli Studi di Napoli “Federico II”.

Durante il periodo di tirocinio magistrale ho lavorato allo sviluppo di un saggio di neutralizzazione sicuro in vitro basato sulla produzione di particelle lentivirali pseudotipizzate con le diverse varianti della spike del SARS-CoV2.

Il progetto di Dottorato mira allo sviluppo di nuove strategie diagnostiche e terapeutiche per il cancro al pancreas.

Ultimi 5 articoli (Scopus)

opensearch:totalResults = 3
opensearch:startIndex = 0
opensearch:itemsPerPage = 3
@role = request
@searchTerms = AU-ID(59963674700)
@startPage = 0

@_fa = true
@ref = self
@href = https://api.elsevier.com/content/search/scopus?start=0&count=25&query=AU-ID%2859963674700%29&apiKey=6ae70c855c11cca26b94ca23c22dcbcf
@type = application/json

@_fa = true
@ref = first
@href = https://api.elsevier.com/content/search/scopus?start=0&count=25&query=AU-ID%2859963674700%29&apiKey=6ae70c855c11cca26b94ca23c22dcbcf
@type = application/json


inizio

@_fa = true

@_fa = true
@ref = self
@href = https://api.elsevier.com/content/abstract/scopus_id/105011770350

@_fa = true
@ref = author-affiliation
@href = https://api.elsevier.com/content/abstract/scopus_id/105011770350?field=author,affiliation

@_fa = true
@ref = scopus
@href = https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=105011770350&origin=inward

@_fa = true
@ref = scopus-citedby
@href = https://www.scopus.com/inward/citedby.uri?partnerID=HzOxMe3b&scp=105011770350&origin=inward

Fibroblasts activated by miRs-185-5p, miR-652-5p, and miR-1246 shape the tumor microenvironment in triple-negative breast cancer via PATZ1 downregulation; Cellular and Molecular Life Sciences; December 2025; DOI: 10.1007/s00018-025-05781-y
prism:url = https://api.elsevier.com/content/abstract/scopus_id/105011770350
dc:identifier = SCOPUS_ID:105011770350
eid = 2-s2.0-105011770350
dc:creator = De Luca G.
prism:publicationName = Cellular and Molecular Life Sciences
prism:issn = 1420682X
prism:eIssn = 14209071
prism:volume = 82
prism:issueIdentifier = 1
prism:pageRange =
prism:coverDate = 2025-12-01
prism:coverDisplayDate = December 2025
prism:doi = 10.1007/s00018-025-05781-y
citedby-count = 1

@_fa = true
affilname = Consiglio Nazionale delle Ricerche
affiliation-city = Rome
affiliation-country = Italy

pubmed-id = 40711489
prism:aggregationType = Journal
subtype = ar
subtypeDescription = Article
article-number = 287
source-id = 13903
openaccess = 1
openaccessFlag = true
value:

$ = all

$ = publisherfullgold

$ = repository

$ = repositoryam

value:

$ = All Open Access

$ = Gold

$ = Green

prism:isbn:

@_fa =
$ =

pii =

inizio

@_fa = true

@_fa = true
@ref = self
@href = https://api.elsevier.com/content/abstract/scopus_id/105017247775

@_fa = true
@ref = author-affiliation
@href = https://api.elsevier.com/content/abstract/scopus_id/105017247775?field=author,affiliation

@_fa = true
@ref = scopus
@href = https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=105017247775&origin=inward

@_fa = true
@ref = scopus-citedby
@href = https://www.scopus.com/inward/citedby.uri?partnerID=HzOxMe3b&scp=105017247775&origin=inward

@_fa = true
@ref = full-text
@href = https://api.elsevier.com/content/article/eid/1-s2.0-S2473952925003817

BH3 mimetic drugs overcome the microenvironment-induced resistance to crizotinib in ALK+ anaplastic large cell lymphoma; Blood Advances; 14 October 2025; DOI: 10.1182/bloodadvances.2024015322
prism:url = https://api.elsevier.com/content/abstract/scopus_id/105017247775
dc:identifier = SCOPUS_ID:105017247775
eid = 2-s2.0-105017247775
dc:creator = Pignataro C.
prism:publicationName = Blood Advances
prism:issn = 24739529
prism:eIssn = 24739537
prism:volume = 9
prism:issueIdentifier = 19
prism:pageRange = 4757-4775
prism:coverDate = 2025-10-14
prism:coverDisplayDate = 14 October 2025
prism:doi = 10.1182/bloodadvances.2024015322
citedby-count = 1

@_fa = true
affilname = Università degli Studi di Napoli Federico II
affiliation-city = Naples
affiliation-country = Italy

pubmed-id = 40601898
prism:aggregationType = Journal
subtype = ar
subtypeDescription = Article
article-number =
source-id = 21100886825
openaccess = 1
openaccessFlag = true
value:

$ = all

$ = publisherfullgold

$ = repository

$ = repositoryam

value:

$ = All Open Access

$ = Gold

$ = Green

prism:isbn:

@_fa =
$ =

pii =

inizio

@_fa = true

@_fa = true
@ref = self
@href = https://api.elsevier.com/content/abstract/scopus_id/105009081646

@_fa = true
@ref = author-affiliation
@href = https://api.elsevier.com/content/abstract/scopus_id/105009081646?field=author,affiliation

@_fa = true
@ref = scopus
@href = https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=105009081646&origin=inward

@_fa = true
@ref = scopus-citedby
@href = https://www.scopus.com/inward/citedby.uri?partnerID=HzOxMe3b&scp=105009081646&origin=inward

Targeting Glioblastoma Stem Cells: A40s Aptamer-NIR-Dye Conjugate for Glioblastoma Visualization and Treatment; Biomolecules; June 2025; DOI: 10.3390/biom15060768
prism:url = https://api.elsevier.com/content/abstract/scopus_id/105009081646
dc:identifier = SCOPUS_ID:105009081646
eid = 2-s2.0-105009081646
dc:creator = Affinito A.
prism:publicationName = Biomolecules
prism:issn =
prism:eIssn = 2218273X
prism:volume = 15
prism:issueIdentifier = 6
prism:pageRange =
prism:coverDate = 2025-06-01
prism:coverDisplayDate = June 2025
prism:doi = 10.3390/biom15060768
citedby-count = 1

@_fa = true
affilname = Università degli Studi di Napoli Federico II
affiliation-city = Naples
affiliation-country = Italy

@_fa = true
affilname = AKA Biotech
affiliation-city = Naples
affiliation-country = Italy

pubmed-id = 40563410
prism:aggregationType = Journal
subtype = ar
subtypeDescription = Article
article-number = 768
source-id = 21100394188
openaccess = 1
openaccessFlag = true
value:

$ = all

$ = publisherfullgold

$ = repository

$ = repositoryam

value:

$ = All Open Access

$ = Gold

$ = Green

prism:isbn:

@_fa =
$ =

pii =

Ultimi 5 articoli (PubMed)

  • Fibroblasts activated by miRs-185-5p, miR-652-5p, and miR-1246 shape the tumor microenvironment in triple-negative breast cancer via PATZ1 downregulation
    on 25 Luglio 2025

    The intricate interplay between epithelial and fibroblast cells within the tumor microenvironment plays a crucial role in driving triple-negative breast cancer progression. This crosstalk involves the exchange of various signaling molecules, including growth factors, cytokines, extracellular matrix components, and extracellular vesicles. Recently, we demonstrated that triple-negative breast cancer extracellular vesicles carry and release a specific combination of miRs, including miR-185-5p,...

  • BH3 mimetic drugs overcome the microenvironment-induced resistance to crizotinib in ALK+ anaplastic large cell lymphoma
    on 2 Luglio 2025

    Resistance to first-line chemotherapies and crizotinib in anaplastic large cell lymphoma (ALCL) represents a significant challenge, often leading to a dismal outcome. Despite recent advancements, the dissection of the intrinsic and extrinsic molecular alterations underlying crizotinib resistance in ALCL is still poorly understood. Here, we transcriptionally unraveled the bidirectional interplay between anaplastic lymphoma kinase (ALK)-driven ALCL (ALK+ ALCL) and stromal cells in the presence of...

  • Targeting Glioblastoma Stem Cells: A40s Aptamer-NIR-Dye Conjugate for Glioblastoma Visualization and Treatment
    on 26 Giugno 2025

    Glioblastoma (GBM) is the most aggressive and challenging brain cancer, in terms of diagnosis and therapy. The highly infiltrative glioblastoma stem cells (GSCs) are difficult to visualize and surgically remove with the current diagnostic tools, which often lead to misdiagnosis and false-positive results. In this study, we focused on a groundbreaking tool for specifically visualizing and removing GSCs. We exploited the specific binding of A40s aptamer to EphA2 for the selective delivery of...